PROBLEM SOLVING IN CLINICAL PRACTICE
Galvanised by a respiratory distress diagnosis
1 Department of Newborn Care, Royal Hospital for Women, Randwick, NSW 2031, Australia
2 School of Womens and Childrens Health, University of New South Wales, Kensington, NSW 2032, Australia
3 Electron Microscope Unit, Concord Repatriation General Hospital, Concord, NSW 2139, Australia
4 Department of Pathology, Sydney Childrens Hospital, Randwick, NSW 2031, Australia
Correspondence to:
Dr Kei Lui, Department of Newborn Care, Royal Hospital for Women, Barker Street, Locked Bag 2000, Randwick, 2031 NSW, Australia; Kei.Lui@SESIAHS.HEALTH.NSW.GOV.AU
| The first 150 words of the full text of this article appear below. |
The third pregnancy of a 38-year old Caucasian G3P2 woman had been unremarkable apart from pregnancy-induced cholestasis and group B streptococcus (GBS) detected on a routine 35-week high vaginal swab and for which intrapartum penicillin was administered. Following induction of labour at 38 weeks gestation for worsening cholestasis, the woman proceeded to a normal vaginal delivery of a live male infant. There was no maternal pyrexia and the liquor was clear. The parents were non-consanguineous and neither the parents nor the other siblings had a history of pulmonary disease.
The baby was born with Apgar scores of 8 and 9 at 1 and 5 min, respectively. He developed respiratory distress almost immediately, with an audible grunt, flaring of the alae nasi and tachypnoea. Oxyhaemoglobin saturation (SpO2) was 82% when breathing room air so the infant was placed in a head box (HB) with fractional inspired oxygen (FiO2) of
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